Advisory · September 2026

Scalp Infections

What they look like, why most scalp complaints are not infections, and when to see a clinician.

Reading time · 6 minutes
Scalp infections

Most scalp complaints are not infections. Itch, flaking, oiliness and tenderness usually trace back to seborrhoeic dermatitis, psoriasis, contact reactions or simple irritation. True infection is less common, and most cases fall into three families: fungal, bacterial, and viral.

The reason this matters is that the three families look different, behave differently, and need different treatment. Confusing one for another wastes time and sometimes makes things worse.

Fungal: tinea capitis and kerion

Tinea capitis is a fungal infection of the scalp caused by dermatophytes — most often Trichophyton and Microsporum species.1 It is predominantly a disease of children. Classic features are patchy hair loss with scale, broken hairs at the surface (“black dots”), and sometimes lymph nodes at the back of the neck.1

A kerion is the inflammatory form: a boggy, tender, pus-studded plaque that can be mistaken for a bacterial abscess.2 The pus is part of the immune response to the fungus, not a separate bacterial problem, and incising it does not help.

The point worth keeping is this: topical antifungal shampoos alone do not cure tinea capitis. Oral antifungal treatment is required, typically griseofulvin or terbinafine, for several weeks.3 Shampoos help reduce shedding of spores to household contacts but they do not reach the hair shaft itself.3

Bacterial: folliculitis, furuncles and impetigo

Bacterial scalp infections most commonly involve Staphylococcus aureus.4 Superficial folliculitis presents as small, tender, pus-tipped bumps around individual hair follicles. A furuncle (boil) is a deeper, more painful nodule. Impetigo, more common in children, shows honey-coloured crusts.5

Most superficial bacterial infections of the scalp respond to topical antibiotics and gentle cleansing.4 Deeper or recurrent infection, fever, or spreading redness warrants oral antibiotics and clinical assessment, particularly given the rise of methicillin-resistant strains.6

Two scalp-specific conditions deserve naming. Folliculitis decalvans is a chronic, scarring form of folliculitis that progressively destroys follicles and leaves smooth patches of permanent hair loss.7 Dissecting cellulitis of the scalp produces deep, interconnected nodules and sinus tracts, more common in young men, and is poorly responsive to simple antibiotics.8 Both are scarring, both need specialist care, and neither resolves with over-the-counter measures.

Viral: herpes simplex and herpes zoster

Viral infections of the scalp are uncommon but worth recognising. Herpes simplex can occur on the scalp, presenting as grouped vesicles on a red base, often painful or tingling before the rash appears.9

Herpes zoster (shingles) involving the scalp follows a nerve distribution, usually one-sided, with pain that may precede the rash by days.10 Early antiviral treatment within 72 hours of rash onset reduces severity and the risk of post-herpetic neuralgia.10 Anyone with a painful, one-sided scalp rash should see a clinician promptly.

What is not an infection

Several common scalp conditions are routinely mistaken for infection. Seborrhoeic dermatitis produces flaking and itch that can mimic mild tinea, but the underlying mechanism is an inflammatory response to commensal Malassezia yeast, not infection.11 Psoriasis causes thicker, silvery scale on well-defined plaques. Contact dermatitis from a new product produces redness and itch without pustules or broken hairs.

The practical signal that something is infectious rather than inflammatory: localised pus, tender swelling, fever, lymph nodes, or a rash that follows a nerve line. Diffuse flaking and itch across the whole scalp, without these features, is almost never infection.12

When to see a clinician

See a clinician for: patchy hair loss with scale in a child; a boggy or pus-filled scalp lump; honey-coloured crusts; a painful one-sided rash; fever with scalp tenderness; or any scalp problem that is worsening despite reasonable care. Scarring conditions, in particular, are time-sensitive. Treatment started early preserves follicles; treatment started late does not bring them back.

Most scalp complaints are not infections, but the ones that are deserve to be named correctly and treated promptly. Guesswork at home is fine for flaking. It is not fine for pus, pain, or hair coming out in patches.

Sources

  1. Hay RJ. Tinea Capitis: Current Status. Mycopathologia. 2017;182(1-2):87–93.
  2. Proudfoot LE, Higgins EM, Morris-Jones R. A retrospective study of the management of pediatric kerion in Trichophyton tonsurans infection. Pediatr Dermatol. 2011;28(6):655–7.
  3. Gupta AK, Drummond-Main C. Meta-analysis of randomized, controlled trials comparing particular doses of griseofulvin and terbinafine for the treatment of tinea capitis. Pediatr Dermatol. 2013;30(1):1–6.
  4. Stevens DL, Bisno AL, Chambers HF, et al. Practice guidelines for the diagnosis and management of skin and soft tissue infections: 2014 update by the IDSA. Clin Infect Dis. 2014;59(2):e10–52.
  5. Hartman-Adams H, Banvard C, Juckett G. Impetigo: diagnosis and treatment. Am Fam Physician. 2014;90(4):229–35.
  6. Liu C, Bayer A, Cosgrove SE, et al. Clinical practice guidelines by the IDSA for the treatment of MRSA infections in adults and children. Clin Infect Dis. 2011;52(3):e18–55.
  7. Otberg N, Kang H, Alzolibani AA, Shapiro J. Folliculitis decalvans. Dermatol Ther. 2008;21(4):238–44.
  8. Scheinfeld N. Dissecting cellulitis (Perifolliculitis Capitis Abscedens et Suffodiens): a comprehensive review focusing on new treatments and findings. Dermatol Online J. 2014;20(5):22692.
  9. Fatahzadeh M, Schwartz RA. Human herpes simplex virus infections: epidemiology, pathogenesis, symptomatology, diagnosis, and management. J Am Acad Dermatol. 2007;57(5):737–63.
  10. Dworkin RH, Johnson RW, Breuer J, et al. Recommendations for the management of herpes zoster. Clin Infect Dis. 2007;44 Suppl 1:S1–26.
  11. Borda LJ, Wikramanayake TC. Seborrheic dermatitis and dandruff: a comprehensive review. J Clin Investig Dermatol. 2015;3(2):10.13188/2373-1044.1000019.
  12. Schwartz RA, Janusz CA, Janniger CK. Seborrheic dermatitis: an overview. Am Fam Physician. 2006;74(1):125–30.
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